One of the major forms of research conducted by criminologists is searching for and analyzing information in published materials. These materials include:
peer-reviewed journal articles (articles published in academic journals)
government documents (reports such as those published by the National Institute of Justice [NIJ], FBI, etc.)
mass media reports (articles in major newspapers [The New York Times, The Washington Post, etc.] and electronic media outlets [ABC, Fox, CNN, etc.])
Each of these kinds of information sources has its strengths and weaknesses. Depending on the kind of research a criminologist is conducting, one of these may prove more useful than the others.
Your mission in this assignment is to find three examples of publications, articles, reports, or stories from each of these kinds of information sources. You will have a total of (9) nine sourcesâ three for each information source type. The topic of the work is to be a specific crime within the Criminal Justice System.
Sample Solution
Chronic myeloid leukemia (CML) remains as a malignant clonal disorder featured by virtue of hyperproliferation of myeloid cells localized in peripheral blood, spleen as well as bone marrow [1]. many cases of chronic myeloid leukemia (CML) are recognized amid blood tests did for another reason. patients with Chronic myeloid leukemia (CML) and BCR-ABL T315I mutation do not profit from treatment with presently accepted tyrosine kinase inhibitors [2]. The aim of the Study to Clone the Gene of interest TROVE2 transcript variant 1 (which involve in CML) that extracted from KCL22 cell line into E.coli with pBluescript plasmid. Introduction: Chronic myeloid leukemia (CML) remains as a malignant clonal disorder featured by virtue of hyperproliferation of myeloid cells localized in peripheral blood, spleen as well as bone marrow [1]. The diagnosis by molecular as well as cytogenetics methods relies on detecting Philadelphia chromosome, featuring reciprocal translocation t(9;22). The consequences of such translocation results in the BCR-ABL oncogene which in turn activates tyrosine kinase that produces abnormal protein activation ending up in deregulation of the hematopoietic stem cells that triggers myeloid cell hyper proliferation ending up to CML. On the other hand, each of the cancerous cell line is being characterized by virtue of intracellular or extracellular biomarkers [2]. The utilization of âautoimmune antigenâ for the treating Chronic Myeloid Leukemia acts as a probable alternative. Variety of autoantibodies from the serum autoimmune patientsâ, are available including those specific antibodies for nuclear antigens. Such antibodies are commonly termed as antinuclear antibodies (ANA). These ANA are indeed specific against autoantigen Ro also known as SS-A else anti-Ro antibodies. These serve as diagnostic marker for detecting the condition. Ro antigen contains two major proteins Ro52 (SSA1, TRIM21, else RNF81) along with Ro60 (TROVE2 else SSA>
Chronic myeloid leukemia (CML) remains as a malignant clonal disorder featured by virtue of hyperproliferation of myeloid cells localized in peripheral blood, spleen as well as bone marrow [1]. many cases of chronic myeloid leukemia (CML) are recognized amid blood tests did for another reason. patients with Chronic myeloid leukemia (CML) and BCR-ABL T315I mutation do not profit from treatment with presently accepted tyrosine kinase inhibitors [2]. The aim of the Study to Clone the Gene of interest TROVE2 transcript variant 1 (which involve in CML) that extracted from KCL22 cell line into E.coli with pBluescript plasmid. Introduction: Chronic myeloid leukemia (CML) remains as a malignant clonal disorder featured by virtue of hyperproliferation of myeloid cells localized in peripheral blood, spleen as well as bone marrow [1]. The diagnosis by molecular as well as cytogenetics methods relies on detecting Philadelphia chromosome, featuring reciprocal translocation t(9;22). The consequences of such translocation results in the BCR-ABL oncogene which in turn activates tyrosine kinase that produces abnormal protein activation ending up in deregulation of the hematopoietic stem cells that triggers myeloid cell hyper proliferation ending up to CML. On the other hand, each of the cancerous cell line is being characterized by virtue of intracellular or extracellular biomarkers [2]. The utilization of âautoimmune antigenâ for the treating Chronic Myeloid Leukemia acts as a probable alternative. Variety of autoantibodies from the serum autoimmune patientsâ, are available including those specific antibodies for nuclear antigens. Such antibodies are commonly termed as antinuclear antibodies (ANA). These ANA are indeed specific against autoantigen Ro also known as SS-A else anti-Ro antibodies. These serve as diagnostic marker for detecting the condition. Ro antigen contains two major proteins Ro52 (SSA1, TRIM21, else RNF81) along with Ro60 (TROVE2 else SSA>
